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dc.contributor.authorAminah, Siti-
dc.contributor.authorPusparini, Iin-
dc.contributor.authorLailiyya, Nushrotul-
dc.contributor.authorGaniem, Ahmad Rizal-
dc.contributor.authorGamayani, Uni-
dc.contributor.authorWibisono, Yusuf-
dc.contributor.authorHuda, Fathul-
dc.contributor.authorSribudiani, Yunia-
dc.contributor.authorAchmad, Tri Hanggono-
dc.date.accessioned2024-12-02T02:01:21Z-
dc.date.available2024-12-02T02:01:21Z-
dc.date.issued2022-
dc.identifier.issn2338-6223-
dc.identifier.urihttp://localhost:8080/xmlui/handle/123456789/8672-
dc.description.abstractSpinocerebellar ataxia (SCA) 3 is a neurodegenerative disease which involves cerebellum and extra cerebellum. Neuropathy in SCA3 manifests in various ways, including axonal and demyelination lesions in sensory and motor nerves. There has not been any study that describes the peripheral neuropathy characteristics of SCA3 patients in Indonesia at the time of this publication. This paper reports a case of a 43-year-old male with known spinocerebellar ataxia 3 presented with hereditary ataxia and mild numbness in both palms since two years before. No abnormalities were found during the sensory examination. The NCS showed severe axonal demyelinating sensorimotor peripheral neuropathy. In magnetic resonance imaging (MRI), an atrophy in the cerebellum with cerebral multiple lacunar infarction was identified. Electrophysiological results revealed profound axonal lesion in peripheral nerves. To conclude, peripheral neuropathy in SCA3 represents the dominance of axonal lesions in motor nerves.en_US
dc.language.isoen_USen_US
dc.publisherMajalah Kedokteran Bandung (MKB)en_US
dc.relation.ispartofseriesResearch Article;57-62-
dc.subjectNCSen_US
dc.subjectperipheral neuropathyen_US
dc.subjectSCA3en_US
dc.titleSpinocerebellar Ataxia 3 (SCA3) Patient with Peripheral Neuropathyen_US
dc.typeArticleen_US
Appears in Collections:VOL 54 NO 1 (2022)

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